15 enero 2015
Defects in the protein Sox10, a transcription factor that regulates gene expression, may play a role in the development of post-operative GI dysfunction in Hirschsprung’s disease patients, according to new research. Hirschsprung’s disease is a congenital disorder caused by the absence of ganglion cells in the colon, which causes problems with passing stool.
http://feeds.sciencedaily.com/~r/sciencedaily/~3/5U7idZ-K2fU/150115111523.htm
